Increased perfusion of the femoral head in transient bone marrow edema syndrome

Kyung Hoi Koo, In Oak Ahn, Hae Ryong Song, Shin Yoon Kim, John Paul Jones

Research output: Contribution to journalArticle

37 Citations (Scopus)

Abstract

The pathophysiology of transient bone marrow edema syndrome is not known. Ischemia has been suggested as the pathophysiologic factor, because the histologic findings are similar to those of early stage osteonecrosis. Angiographic studies of osteonecrotic femoral heads have shown arterial interruption and impaired perfusion. The current report describes the angiographic and scintigraphic findings of transient bone marrow edema syndrome of the hip in a 45-year-old man. The nutrient arteries were dilated, and the femoral head perfusion was increased compared with the unaffected contralateral side. These findings suggest that a vasomotor response plays a role in the pathogenesis of tranease might be a reversible process after temporary ischemia of the femoral head.

Original languageEnglish
Pages (from-to)171-175
Number of pages5
JournalClinical Orthopaedics and Related Research
Issue number402
DOIs
Publication statusPublished - 2002 Sep
Externally publishedYes

ASJC Scopus subject areas

  • Surgery
  • Orthopedics and Sports Medicine

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